Subject:

Ch20: Abdominal Injury(腹部损伤)

Preparatory Mindset

Abdominal injury = closed (blunt) & open (penetrating) trauma to the abdomen. The lecture (Dr. Pan, 13.4KB) covers the principles of first aid, early diagnosis, and treatment; the differential of liver, spleen, pancreas & bowel rupture; and the diagnostic steps for closed injury. This pairs with the Trauma chapter (Ch12) — the ATLS/FAST approach applies here specifically to the abdomen.

Exam mindset: solid organs (liver, spleen) → hemorrhage; hollow organs (bowel) → peritonitis; the FAST scan & unstable → laparotomy vs stable → CT decision; and the classic signs (Kehr's sign for spleen, peritonitis for bowel).

Core Concepts

1. Overview & classification (MUST KNOW)

- Open (penetrating) injury (开放性): stab, gunshot — wound present, easier to recognize; exploration needed (may be laparotomy) - Closed (blunt) injury (闭合性): no wound — harder to diagnose (hidden visceral injury); RTA, falls, assault - Iatrogenic injury: endoscopy, surgery

2. Mechanisms of injury (MUST KNOW)

- Spleen (most commonly injured solid organ in blunt trauma) — hemorrhage - Liver (second) — hemorrhage, bile leak - Kidney — hematuria - Pancreas — delayed presentation (retroperitoneal) - Small bowel (hollow — most common hollow organ) — perforation → peritonitis - Colon, mesentery, diaphragm, bladder

3. Clinical manifestations (MUST KNOW)

- Spleen: left upper quadrant pain, Kehr's sign (left shoulder-tip pain — blood irritates the diaphragm, phrenic C3-5), hypotension, tachycardia, falling Hb; left lower rib fractures - Liver: right upper quadrant pain, referred right shoulder, hypotension; hemobilia rare - Kidney: flank pain, hematuria, flank bruising

- Bowel perforation: progressive abdominal pain, guarding, rigidity, rebound tenderness, absent bowel sounds, fever, tachycardia; free air (pneumoperitoneum) — later - Pancreas: epigastric pain radiating to the back (delayed, retroperitoneal — easily missed); ↑ amylase/lipase; periumbilical (Cullen) / flank (Grey-Turner) ecchymosis (late, also pancreatitis)

4. Diagnosis (MUST KNOW)

5. Treatment principles (MUST KNOW)

- Hemodynamic instability + positive FAST (or peritonitis) - Evisceration, impalement - Penetrating injury with peritonitis/shock (gunshot wounds usually explored) - Free intraperitoneal air - Deteriorating/ongoing bleeding despite resuscitation - Evidence of hollow viscus perforation (peritonitis, gas)

- Spleen: splenorrhaphy or splenectomy (unstable); vaccinate (encapsulated organisms: pneumococcus, meningococcus, Hib) & lifelong antibiotics/post-splenectomy sepsis awareness after splenectomy - Liver: packing & Pringle maneuver (control porta hepatis), hepatorrhaphy, angioembolization; segmental resection; major vascular control - Kidney: NOM for minor; nephrectomy for non-salvageable (check contralateral) - Bowel: repair (primary closure) or resection & anastomosis; consider stoma if peritonitis/severe contamination/hemodynamic instability - Pancreas: drain; distal pancreatectomy for body/tail injury; ERCP/stenting for duct injury - Bladder/ureter: repair + drainage

High-Yield Points

Topic Summary

Abdominal injury: open (penetrating) & closed (blunt). Solid organs (spleen most common, liver, kidney, pancreas) bleed → hypovolemic shock; hollow organs (small bowel most common) perforate → delayed peritonitis. Classic signs: Kehr's (spleen), free air (hollow viscus), hematuria (kidney), Cullen/Grey-Turner (pancreas, late). Diagnosis: ABCDE + FAST (unstable + positive → laparotomy) + CT (stable → definitive imaging, NOM for low-grade liver/spleen) + serial exams. Treatment: resuscitate; laparotomy for instability/peritonitis/evisceration/free air; non-operative management with angioembolization for stable solid-organ injuries; surgical principles (splenectomy + vaccination, liver packing/Pringle, bowel resection, pancreatic drain/resection); damage control in the unstable; broad-spectrum antibiotics & tetanus prophylaxis.

LMCHK OSCE Practice